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These conclusions posture EBNA-3a just like a vital modulator of B cellular proliferation and survival during EBV latency, and as a applicable CD8⁺ T cell aim in the two very simple immunological experiments and translational examine.
EBNA-3a appears to control a subset of cellular genes, a number of which overlap with People regulated by EBNA2. The two proteins can act antagonistically or cooperatively to manage gene expression. These conclusions posture EBNA-3a for a essential modulator of B cell proliferation and survival through EBV latency, and being a pertinent CD8⁺ T cell target in both primary immunological scientific tests and translational study.
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